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EXOPHTHALMIC GOITER, NODULAR TYPE

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  1. EXOPHTHALMIC GOITER

 

 

Mrs. M. J., age 35, in July, 1943, showed rapidly developing weakness, tremor, sweating, great changes in appearance, extreme exophthalmus, rapid loss of weight and strength, excitement, excessive nervousness, tremor, jerking of the muscles, spasms with toes bending inward, and use of the fingers became difficult. The thyroid region was enlarged by a number of hard nodular tumors, rapidly increasing, pulse thready and too rapid to count accurately, blood pressure 190/110, and B.M.R. plus 104%. She was too weak to walk and had to be carried into Dr. Julian Baldor’s office on November 10th, 1943, after being under iodine therapy, ice bags to the neck, and absolute quiet from July to November 10th, while steadily deteriorating. She thought she was losing her mind, had hallucinations. She was given 2 micro micrograms of the serial system of Carbonyl groups in the triceps muscle. In two weeks, a remarkable change for the better was evident. She was stronger, could sleep, gained weight, etc. In twelve weeks, she was completely normal, physically and symptomatically, B.M.R. plus 6%, pulse 80, blood pressure 140/80, back to normal weight and strength, working hard by carrying a suitcase weighing 50 pounds in and out of houses as a demonstrator, playing in an orchestra, etc. Eyes, thyroid, and ner­vous system completely normal and have remained so to date.

 

 

Comment: In this case, somewhere in the body, because of block of FCG function, important cells could not obtain the energy of ATP; they were starved of energy. So the hormone message was sent to the thyroid to whip up all the tissues to produce more ATP to overcome the energy starvation of that group of cells where ATP energy was blocked from entering the working mechanism. Thereby, the thyroid gland was stimulated to the limit and the tissues were depleted to utter exhaustion. The Carbonyl compound of high oxidation potential caused removal of the obstructing toxin, as we have explained. As soon as the FCG of energy reception was freed and could go back to work, the tissue that was starved received all the energy it could use, so the call on the thyroid ceased and its nodular hyperplasia and activity sub­sided to normal. The depleted tissues were no longer forced to produce ATP and all was well.

 

 


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