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THYMUS GLAND DEFICIENC AND MUSCULAR DYSTROPHIES

The patterns of endocrine deficiency vary in some instances from that exposed in the exophthalmic goiter case, Mrs. M. J. Here, the deficiency was not in the thyroid gland that attracted so much attention, but in some other tissue that could not accept the energy of ATP into its funct­ional units and hence, was starving for energy. So some nerve or hormonal factor acted on the thyroid to produce thyroxin to whip up the tissues to produce energy carried as ATP to supply the starving tissue. However, the block to the FCG of energy acceptance for function prevented this energy from being used and a vicious circle was established that was leading to fatal exhaustion. After the FCG was freed so it could accept the energy, the whole mischief was normalized.

In the muscular dystrophies, the thymus is the essential deficient tissue upon which the muscle deficiency depends. In both the thyroid and in the complete thymus deficiencies, the inability to accept energy into the functional mechanisms is evident in the hyperplasia of the gland, and the increased use of oxygen and higher basal metabolism rate. In the thyroid case, the BMR was as high as 104%, but in complete thymus deficiency cases it is very much less elevated, though enough to indicate the inability to use the energy of ATP. It is also evident that the thymus defect may not be complete, but may depend on the inability to use its specific trace element, manganese, as a thyroid case may not use iodine, or an anemia case may not use cobalt. So one must provide a concept of how the thymus gland works as we have for diabetes, especially, because orthodoxy has no solution.

There are a few facts that can be organized for a practical pattern of its function. Alpha tocopherol is essential to its function as well as manganese. The spent product of tocopherol appearing in the urine is in the form of the hydroquinone of tocopherol Therefore, our Thesis is simply that tocopherol is oxidized to the quinone, which on performing its task, is reduced to the hydro­quinone. In other words, the manganese is used by the thymus Hassall’s cells as a co-factor, possibly as the trioxide, to oxidize tocopherol to its quinone and the quinone serves as an oxidizing agent (hydrogen or electron acceptor) in the further function of the gland, as in the production of a substance for the devel­opment and function of the muscles, and of the reproductive system. In this latter function, the use of ATP is required and when the FCG of energy accept­ance of the Hassall’s cells cannot accept this energy because of a block via an integrated pathogen; thus the thymus and muscle deficiencies are complete until the block is removed. Many years ago we used the serial system of Carbonyl groups and those as activated in Benzoquinone, to accomplish the liberation of the FCG.

But the deficiency in the thymus may not be complete and may involve the simple oxidation of manganese to its trioxide. The supply of fair but non-toxic amounts of manganese to the tissues in general Josephson (8) found would correct such cases. Evidently the chain of subsequent processes was unimpeded and the body cells in general oxidized the manganese. But when the FCG of energy acceptance is integrated with a pathogen, the use of the Reagent given to the thyroid cases, is also required to reverse the disease picture in Parkinson’s disease, myasthenia gravis, and some other forms of muscular dystrophy, as reported by our collaborators. We will give a special discussion to this subject with photographs demonstrating muscle reconstruction and return of function.

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