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PARALYTIC DOG DISTEMPER
While rabies is a 100% fatal example of the lytic type of viral integration, paralytic distemper is a 100% fatal expression of the symbiotic type. It has responded with 100% restoration of function in 90% of cases treated by the Oxidation and the Reduction procedures, outlined here in private practice. In the Treatment of all cases that came along, with care left to the owner, the Army Hospital for small animals secured an 80% recovery rate in two hundred cases in all types of distemper. (‘“ Veterinaria,” Vol. IV, No. 1, p. 21, 1950, Colombo and Carneiro.) (12)
A typical case is that of the pointer, Singe, age 10 years. He was treated with the Oxidation Reagent on October 14, 1960, after he had become paralyzed in the right half of the torso and the left back quarter as the pictures show. The prodromal symptoms of trembling, loss of appetite, and sadness, lasted two weeks and the paralysis was of two weeks duration, before the Treatment was given. There was visible atrophy of all muscles concerned. The veterinarian offered to sacrifice the dog, as it was a hopeless case. The recovery took two weeks to overcome the paralysis and in two more weeks the atrophy was repaired as well, and the dog perfectly well. This was an early case and the reversal of the pathogenesis took as long as its development. Cases created with the Reduction Reagent showed no difference in their recovery percentages or course. Restoration of function of the paralyzed milk producing cells takes twenty-four hours after either Reagent is used in cows treated for hoof and mouth disease. (13)
CANCER
Warburg’s Thesis on tissue oxidation is well known and his report on anoxia as the etiological factor in cancer (14) demonstrates this fact. He, however, does not explain how anoxia produces cancer and concludes that the pathology is irreversible. Our Thesis shows the essential place of anoxia in the pathogenesis, as it provides for the integration of a co-factor the pathogen or virus, with the host cell’s functional mechanism via free radical additions or azomethine condensations.We have demonstrated for decades that the pathogen can be separated from the host cell by the two mechanisms of cleavage — one at the position alpha to the activating double bond, and the other by cleavage through the double bond itself. Excess tissue of the tumors is placed in a position for normal function, but being in excess, it is digested and absorbed like a blood clot, and thereby serves as nutrition. (Koch, Cancer Journal, October, 1924). The pathogenesis is thus reversed!
Warburg suggested that the mitogenic energy comes from glycolysis and it seems reasonable. However, our experience allows us to conclude that it arises in the polymerization of incompletely combusted metabolites (produced by germs trapped in an anoxic scar) that have entered the host cell and integrated with the mitotic mechanism, or by the energy arising in the polymerization or a part of a provirus that has integrated with the host cell’s mitotic mechanism.We have observed in our earliest experience that when cancer is given small, rapidly polymerizing, unsaturated free radicals, their growth is terrifically stimulated by the energy, so liberated. Whereas, if a large, inert, free radical is given, their growth ceases and involution sets in. Here polymerization has been terminated and the source of energy is cut off. Synthetic carcinogens serve as the initiators of the co-polymerization of the bacterial metabolite or provirus during anoxia, when the free radical formed by its dehydrogenation at the hands of the FCG adds to one pole of the double bond that activates the FCG, and the free radical thus formed at the other pole adds to the unsaturated ethylenic linkage of either pathogen, thereby producing a free radical that continues the polymerization, as an end to end process. Very little energy is required for mitosis, and the energy liberated by slow polymerization should be sufficient, especially as it is liberated in the energy generating mechanism. The removal of each pathogen involved requires oxygen and an efficient dehydrogenator, as we explained earlier.
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