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THE INTEGRATION OF PATHOGEN AND HOST CELL CRITICAL ATOMIC GROUPS AND THEIR SEPARATIONS

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  8. Now match the patients’ descriptions of their symptoms (1-7) with the medical terms (a-g).
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  10. Read and learn the following words and their equivalents.

(Schematic)

 

 

To rupture the integration oxidatively, the Therapy dehydrogenator re­moves H(3) of the initiating pathogen producing a free radical that adds molecular oxygen to become a peroxide free radical that cleaves C(4) from C(6) producing a Carbonyl group at the latter. C(4) also becomes a Carbonyl group which being positive remains attached to the negative C(13). By gaining a Carbonyl group, the pathogen looses its parasitism and becomes autonomous.

 

The polymerization bond between C(14) of the host’s FCG activat­ing double bond and C(7) of the pathogen’s invites cleavage as C(14), is positive in polarity and tends to release its H atom to the action of a dehydrogenator of appropriate qualities, as offered in the Therapy Reagent. A free radical is formed there and a peroxide free radical results in the presence of oxygen that cleaves C(14) from C(7) of the pathogen, forming two terminal Carbonyl groups. The Functional System of the host cell thus now holds a cluster of three Carbonyl groups to serve its dehydrogenating function as activators and as dehydrogenators. This is a quite formidable array, via its orbital-mechanics. The Carbonyl group won by the pathogen attracts electrons from the methylene group alpha to it and thus releases its hydrogen atom to any dehydrogenator at hand, as the cytochrome or ferrous-ferric electron acceptor systems, and so a new Carbonyl group is formed at each terminal again; a process that can be repeated until the pathogen is burned out of the way.

 

 

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